cigarette smoke increases mitochondrial ros lung epithelial cells Cigarette Smoke-Induced Reactive Oxygen Species Formation: A Concise Review

SS-31 mitigates oxidative stress and restores mitochondrial function in cigarette smoke-damaged oral epithelial cells via PINK1-mediated mitophagy

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Sources and production of ROS in the lung. Activation of macrophages,

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The Convergent Immunopathogenesis of Cigarette Smoke Exposure: From Oxidative Stress to Epigenetic Reprogramming in Chronic Disease

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Role of Lysocardiolipin Acyltransferase in Cigarette Smoke-Induced Lung Epithelial Cell Mitochondrial ROS, Mitochondrial Dynamics, and Apoptosis | Cell Biochemistry and Biophysics

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Figure 1 from Lung Injury and Lung Cancer Caused by Cigarette Smoke-Induced Oxidative Stress: Molecular Mechanisms and Therapeutic Opportunities Involving the Ceramide-Generating Machinery and Epidermal Growth Factor Receptor

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Prolonged cigarette smoke exposure alters mitochondrial structure and function in airway epithelial cells | Respiratory Research

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Cigarette smoke causes a bioenergetic crisis in RPE cells involving the downregulation of HIF-1α under normoxia

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